Description
Functional Bowel Disorders (FBD), including Irritable Bowel Syndrome (IBS), are highly prevalent gastrointestinal conditions classified as Disorders of Gut-Brain Interactions (DGBIs) under the Rome IV criteria. These conditions, marked by abdominal pain, altered bowel habits, and significant psychological comorbidities, present substantial challenges in both diagnosis and treatment. Conventional therapies primarily target gut-centric mechanisms but often yield limited efficacy, underscoring the need for approaches that account for the complex interplay of the gut-brain axis in DGBI pathophysiology.
Disciplines
Gastroenterology | Internal Medicine | Investigative Techniques | Neurosciences | Pain Management | Translational Medical Research
Document Type
Poster
Recommended Citation
Draper, Lauren; Pfister, Devin; and Zaugg, Ethan, "Transcranial Magnetic Stimulation as a treatment modality for Functional Bowel Disorder and Irritable Bowel Syndrome and possible correlations with the Gut-Brain Axis: A review" (2025). Annual Research Symposium. 13.
https://ecommons.roseman.edu/researchsymposium/2025/clinical/13
Included in
Gastroenterology Commons, Internal Medicine Commons, Investigative Techniques Commons, Neurosciences Commons, Pain Management Commons, Translational Medical Research Commons
Transcranial Magnetic Stimulation as a treatment modality for Functional Bowel Disorder and Irritable Bowel Syndrome and possible correlations with the Gut-Brain Axis: A review
Functional Bowel Disorders (FBD), including Irritable Bowel Syndrome (IBS), are highly prevalent gastrointestinal conditions classified as Disorders of Gut-Brain Interactions (DGBIs) under the Rome IV criteria. These conditions, marked by abdominal pain, altered bowel habits, and significant psychological comorbidities, present substantial challenges in both diagnosis and treatment. Conventional therapies primarily target gut-centric mechanisms but often yield limited efficacy, underscoring the need for approaches that account for the complex interplay of the gut-brain axis in DGBI pathophysiology.